This standard specifies the diagnostic criteria and treatment principles for occupational acute phosphine poisoning. This standard applies to the diagnosis and treatment of phosphine poisoning in occupational activities. GBZ 11-2002 Occupational acute phosphine poisoning diagnostic criteria GBZ11-2002 Standard download decompression password: www.bzxz.net

1CS13.100
National Occupational Health Standard of the People's Republic of China GBZ 11—2002
Diagnostic Criteria of Occupational Acute Phosphine Poisoning2002-04-08 Issued
Ministry of Health of the People's Republic of China
Implementation on 2002-06-01
Article 5.1 of this standard is recommended, and the rest are mandatory. This standard is formulated in accordance with the "Law of the People's Republic of China on the Prevention and Control of Occupational Diseases". From the date of implementation of this standard, if there is any inconsistency between the original standard GB7797-1987 and this standard, this standard shall prevail. Acute carbon tetrachloride poisoning may occur in occupational activities involving exposure to phosphine. In order to protect the health of those exposed to phosphine, facilitate the prevention and control of poisoning, and in accordance with the requirement that the diagnostic standards for occupational diseases should reflect the latest clinical progress, GB7797-1987 has been revised.
The original standard stipulates the diagnostic criteria and treatment principles for occupational acute phosphine poisoning. The relevant literature and data at home and abroad in the past decade show that the relevant provisions of the diagnostic indicators of phosphine poisoning involving damage to important organs such as the heart, lungs, brain, and kidneys can be implemented according to the principle of the same manifestation of target organ damage of occupational diseases, and can refer to the relevant provisions in the relevant general standards. The appendix of this standard adds an explanation of the relationship between this standard and the relevant general standards. Appendix A of this standard is an informative appendix.
This standard is proposed and managed by the Ministry of Health of the People's Republic of China. This standard was drafted by the Occupational Health and Poisoning Control Institute of the Chinese Center for Disease Control and Prevention and the Central Hospital of Bailu Chemical Fiber Group Company in Henan Xiang. The participating units include Xi'an Central Hospital and Xinxiang Institute of Occupational Disease Prevention and Control in Henan Province. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic Criteria for Occupational Acute Hydrogen Poisoning
GBZ11-2002
Acute phosphine poisoning is a systemic disease caused by inhalation of high concentrations of phosphine gas, mainly with damage to the nervous system and respiratory system.
1 Scope
This standard specifies the diagnostic criteria and treatment principles for occupational acute phosphine poisoning. This standard applies to the diagnosis and treatment of phosphine poisoning in occupational activities. 2 Normative references
The clauses in the following documents become clauses of this standard through reference in this standard. For any dated referenced document, all subsequent amendments (excluding errata) or revisions are not applicable to this standard. However, parties to an agreement based on this standard are encouraged to study whether the latest versions of these documents can be used. For any undated referenced document, the latest version shall apply to this standard.
GB/T16180
3 Diagnostic principles
Evaluation of the degree of disability caused by work-related injuries and occupational diseases Diagnostic criteria for occupational toxic liver disease
Diagnostic criteria for occupational acute chemical poisoning respiratory disease Diagnostic criteria for occupational acute chemical poisoning heart disease Based on the occupational history of exposure to high concentrations of phosphine, the onset is relatively fast. Combined with clinical symptoms, signs and other necessary clinical examinations, reference to the results of on-site labor hygiene investigations, comprehensive analysis, and exclusion of other diseases with similar symptoms, a diagnosis can be made 4 Observation subjects
Have headaches, fatigue, nausea, coughs and other nervous system and respiratory system symptoms, but the symptoms are fewer and milder, and they usually disappear within 24 hours after breaking away from contact.
5 Diagnosis and classification standards
5.1 Mild poisoning
One of the following conditions:
a) Mild impaired consciousness;
b) Mild dyspnea, a small amount of dry and wet rales heard in the lungs, consistent with chemical bronchitis or peribronchitis. 5.2 Severe poisoning
In addition to the symptoms of mild poisoning, one of the following conditions is present, or one of the following conditions is present at the beginning of poisoning: a) Coma, convulsions;
b) Pulmonary edema;
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shock;
Obvious myocardial damage;
Obvious liver and kidney damage.
Treatment principles
Treatment principles
6.1.1 Immediately leave the scene and keep quiet. 6.1.2 The subject should be observed for 24 hours and treated according to the situation. Poisoned patients should stay in bed and be observed for at least 24 to 48 hours to detect changes in their condition early.
6.1.3 Treatment is mainly symptomatic and supportive. Coma, pulmonary edema, myocardial or liver and kidney damage should be treated early and actively. The treatment principles and nursing are the same as those of internal medicine. 6.2 Other treatments
6.2.1 Poisoned patients can generally be cured. Mild poisoning patients usually recover within 1 to 2 weeks and can resume their original work after recovery. Severe poisoning patients can also fully recover after active treatment. 6.2.2 Reexamination A small number of patients still have obvious symptoms after rescue and after the acute phase, and they can be treated as appropriate according to the examination results. Instructions for the correct use of this standard
See Appendix A (Informative Appendix).
..comAppendix Abzxz.net
(Informative Appendix)
Instructions for the correct use of this standard
A.1 This standard applies to acute phosphine poisoning. Substances that can produce phosphine include calcium phosphide, aluminum phosphide, zinc phosphide, etc. Elemental phosphorus can only produce phosphine under specific reducing conditions. The effects of phosphorus, other inorganic compounds and organic compounds of phosphorus on humans are different from those of phosphine. The diagnosis and treatment of poisoning are also different. A.2 The manufacturing, packaging, transportation of aluminum phosphide and zinc phosphide, and the use of aluminum phosphide to fumigate grains, furs and other industries can be exposed to higher concentrations of phosphine. Acetylene gas manufacturing and ferrosilicon transportation operations will also produce phosphine due to the mixing of impurities such as calcium phosphide in the raw materials. Workers engaged in these jobs can also be exposed to higher concentrations of phosphine under certain conditions. A.3 The membrane reading of phosphine is about 4.2mg/m2 (3ppm), and poisoning can occur after being exposed to air containing phosphine concentrations of more than 10mg/m2 (7ppm) for several hours. Therefore, the contact person can generally provide a history of contact with a garlic-like odor. On-site phosphine concentration detection is helpful for diagnosis.
A.4 There are no specific manifestations of phosphine poisoning, but according to the comprehensive analysis of the contact situation and clinical examination findings, the diagnosis is generally not difficult.
A.5 Contact reaction does not belong to the scope of poisoning. However, the condition changes relatively quickly after poisoning, so those who have contact reactions should be observed to detect changes in the condition early.
A.6 There is no specific antidote for phosphine poisoning, and drugs cannot be used in emergency treatment. For critically ill patients such as pulmonary edema, if early detection and early treatment can be achieved, the prognosis is better. A.7 The specific manifestations of "lung, heart, liver, kidney..." damage mentioned in the poisoning diagnosis classification standard are not specified. When the standard provisions are actually applied, the relevant general standards plus the diagnosis of acute chemical poisoning respiratory diseases (GBZ73), the diagnosis of heart disease (GBZ74), the diagnosis of toxic liver diseases (GBZ59) and other standards should be implemented. ..com
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