This standard specifies the diagnostic criteria and treatment principles for occupational acute ethylene dichloride poisoning. This standard applies to the diagnosis and treatment of acute and subacute poisoning caused by exposure to ethylene dichloride in occupational activities. GBZ 39-2002 Occupational acute 1,2-dichloroethane poisoning diagnostic criteria GBZ39-2002 Standard download decompression password: www.bzxz.net

1CS13.100
National Occupational Health Standard of the People's Republic of China GBZ39—2002
Diagnostic Criteria of Occupational Acute L, 2-Dichloroethane PoisoningPublished on April 8, 2002
Implemented on June 1, 2002
Ministry of Health of the People's Republic of China
Article 6.1 of this standard is recommended, and the rest are mandatory. This standard is formulated in accordance with the Law of the People's Republic of China on the Prevention and Control of Occupational Diseases. From the date of implementation of this standard, if there is any inconsistency between the original standard GB11506-1989 and this standard, this standard shall prevail. Occupational activities involving exposure to 1,2-dichloroethane (hereinafter referred to as ethylene dichloride) can cause acute poisoning (including subacute poisoning). In order to protect the health of those exposed to it, the state promulgated GB11506-1989 "Occupational Acute 1,2-Dichloroethane Poisoning Diagnostic Criteria and Treatment Principles" in 1989. This standard is linked to GBZ71 and GBZ76 in terms of the diagnostic system. In combination with the research progress in recent years, the diagnostic standard highlights the impact of ethylene dichloride on the central nervous system, especially the toxic encephalopathy caused by severe poisoning, and has been modified in terms of the clauses. Appendix A of this standard is an informative appendix.
This standard is proposed and managed by the Ministry of Health of the People's Republic of China. This standard was drafted by the Guangdong Provincial Institute of Occupational Disease Prevention and Control and the Shenzhen Municipal Health and Epidemic Prevention Station, and the Shenzhen Bao'an District Health and Epidemic Prevention Station and the Zhuhai Municipal Health and Epidemic Prevention Station participated in the drafting.
This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic Standard for Occupational Acute 1,2-Dichloroethane Poisoning GBZ39-2002
Occupational acute dichloroethane poisoning is a systemic disease caused by short-term exposure to high concentrations of dichloroethane in occupational activities, with damage to the central nervous system as the main cause, and may cause liver and kidney damage. 1 ScopeWww.bzxZ.net
This standard specifies the diagnostic criteria and treatment principles for occupational acute dichloroethane poisoning. This standard applies to the diagnosis and treatment of acute and subacute poisoning caused by exposure to dichloroethane in occupational activities. 2 Normative References
The clauses in the following documents become clauses of this standard through reference in this standard. For all dated referenced documents, all subsequent amendments (excluding errata) or revisions are not applicable to this standard. However, parties to an agreement based on this standard are encouraged to study whether the latest versions of these documents can be used. For all undated referenced documents, the latest versions are applicable to this standard.
GB/T16180
3 Diagnostic principles
Diagnostic criteria for occupational toxic liver disease
Diagnostic criteria for occupational acute chemical toxicity nervous system diseaseDiagnostic criteria for occupational acute chemical toxicity nephropathyIdentification of the degree of disability caused by work-related injuries and occupational diseases of employeesAccording to the occupational history of short-term exposure to high concentrations of dichloroethane and the clinical manifestations mainly of central nervous system damage, combined with on-site labor hygiene investigations, comprehensive analysis, and exclusion of similar diseases caused by other causes, a diagnosis can be made. 4 Contact reactions
After short-term exposure to high concentrations of dichloroethane, central nervous system symptoms such as dizziness, headache, and fatigue may occur, which may be accompanied by nausea, vomiting, or eye and upper respiratory tract irritation symptoms, which disappear after a short time after the contact is broken. 5 Diagnosis and classification standards
Mild poisoning
In addition to the aggravation of the above symptoms, one of the following manifestations occurs: a) gait mites:
b) Mild consciousness disorder, such as confusion, drowsiness, and haziness; c) Mild toxic liver disease;
d) Mild toxic nephropathy.
2 Severe poisoning
One of the following manifestations occurs:
a) Moderate or severe consciousness disorder;
b) Grand mal seizure-like convulsions:
c) Focal brain damage, such as cerebellar ataxia, etc.; d) Moderate or severe toxic liver disease
Treatment principles
Treatment principles
6.1.1 On-site treatment: The poisoned person should be quickly removed from the scene, moved to a place with fresh air, and the contaminated clothes should be changed, the contaminated skin should be washed, and the person should be kept warm and closely observed. 6.1.2 Those who have been exposed should be closely observed and given symptomatic treatment. 6.1.3 Focus on the prevention and treatment of toxic encephalopathy, actively treat cerebral edema and reduce intracranial pressure. 6.1.4 There is no specific antidote, and the treatment principles and nursing are the same as those of neurology and internal medicine. 2 Other treatments
Mildly poisoned people can resume their original work after recovery. Severely poisoned people should be transferred away from dichloroethane work after recovery, and those who need labor capacity assessment should be treated in accordance with GB/T16180.
Instructions for the correct use of this standard
See Appendix A (Informative Appendix)
Appendix A
(Informative Appendix)
Instructions for the correct use of this standard
A.1 This standard applies to acute and subacute poisoning caused by exposure to dichloroethane in occupational activities. This standard can also be used as a reference for poisoning caused by exposure to dichloroethane in non-occupational activities. Ethylene dichloride is often used as a raw material for chemical synthesis, industrial solvent, degreasing agent, metal cleaning agent and adhesive, etc. A.2 Acute and subacute ethylene dichloride poisoning mainly manifests as damage to the central nervous system. In particular, subacute poisoning has been the main form of disease in China in the past decade. It is seen in patients who inhale high concentrations of poisoning for a long time. Its characteristics: long incubation period, a few days to dozens of days; insidious onset, the condition may suddenly worsen; clinical manifestations are mainly toxic encephalopathy, with prominent manifestations of cerebral edema, and some severely poisoned patients may have manifestations of focal brain damage, such as cerebellar ataxia. The classification of mild, moderate and severe consciousness disorders is based on Appendix D in GBZ76.
A.3 In acute ethylene dichloride poisoning encephalopathy, brain edema can last for about two weeks, and may recur or suddenly worsen. Treatment should focus on preventing and treating cerebral edema and reducing intracranial pressure, emphasizing "close observation, early detection, timely treatment, and prevention of recurrence". And the treatment observation time should generally not be less than two weeks. A.4 In acute dichloroethane poisoning, clinical observations show liver and kidney damage, but this is more common in people who are poisoned orally. Severe liver and kidney damage caused by occupational poisoning is very rare, especially the kidney. For the classification of toxic liver disease and toxic kidney disease, please refer to GBZ59 and GBZ79.
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